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In isolated rat aorta preparations, the peptide produced concentration-dependent vasodilation that was endothelium-dependent and abolished by L-NAME, and co-immunoprecipitation work traced the response to phosphorylation of Src, caveolin-1, and endothelial nitric oxide synthase, with reduced binding between caveolin-1 and eNOS [1]

Digestive Problems: Increased risk of stomach cancer and gastric polyps

Ideal supporting ingredients complement copper peptide function without creating conflicts

Mechanism-based protocol design Identify primary pathology: Glaucoma primarily ganglion cell death, choose neuroprotective peptides (Cerebrolysin)

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