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In breast cancer cells, a high level of miR-378* induces the metabolic shift from an oxidative to a glycolytic bioenergetics pathway by inhibiting the expression of two PGC-1 partners, ERR (estrogen-related receptor gamma) and GABPA (GA binding protein transcription factor, alpha subunit)
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Consistently, Rho0 AML cells, lacking functional electron transport chain (ETC), were found to be highly sensitive to mitochondrial lipid peroxidation and ferroptosis compared to control cells
Pierce the skin with the needle and inject the B12 into the cat
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