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A number of clinical observations suggest that Down patients may have a functional folate deficiency despite having normal plasma levels of folate and vitamin B 12 [13]

We recommend that all hits should be resynthesised and purified to confirm activity but if this is not possible then compounds from commercial sources should be purified with metal scavenging technology before confirming activity

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These proteins have a substantial influence on the multifaceted functions of nearly all cellular processes

These findings directly support our hypothesis by linking SLC25A40-dependent mtGSH availability to ETC stability and cytokine production in macrophages