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ALS, as excess glutamate accumulation can damage the nerve cells, leading to the progressive disease
We identify a previously unrecognized metabolic reprogramming of ILC2s induced by melatonin, characterized by diversion of glucose and mitochondrial metabolism toward glutathione synthesis
2022;7:e156802
Bone marrow stroma-derived PGE2 protects BCP-ALL cells from DNA damage-induced p53 accumulation and cell death
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