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(7) This stage may go on for decades before enough damage is done, and before one can detect a change in thyroid hormone levels on a blood test at this stage, the TSH, Free T3, and Free T4 levels will be normal
Although biochemically separated from the GSH system, the Trx system shares NADPH as a common reducing cofactor and serves as an alternative pathway for reducing disulfide bonds, making this system especially relevant if the GSH system has been compromised or rendered dysfunctional ( Under oxidative stress these three systems work in concert to detoxify ROS and lipid peroxides, maintain protein structure and function via thiol repair, prevent activation of redox-sensitive inflammatory pathways, and preserve mitochondrial membrane integrity and prevent apoptosis
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*Cytokine regulation: GSH modulates the production of pro-inflammatory cytokines including IL-1, IL-6, TNF-, and IFN- - all of which are elevated in Hashimoto's thyroiditis and Graves' disease B-cell antibody production: Oxidative stress promotes B-cell activation and autoantibody production
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