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Researchers may evaluate: Extracellular-matrix genes Protease-associated genes Protease-inhibitor genes Oxidative-response genes Inflammation-associated pathways Cell-cycle genes Metal-homeostasis genes Stress-response pathways Transcriptomic changes should not be described as proof of tissue renewal, rejuvenation, repair, or anti-aging activity
When NNMT is highly active, it draws on SAM and can lower available NAD
doi: 10.1016/j.jsbmb.2015.02.004 187 FrumanDARommelC
75 For example, in prostate cancer, loss of the histone methyltransferase KMT2C represses ASPP2 via impaired enhancer activity
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