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[32]Du X, Zhu YG, et al
presented a heterozygous CHAC1 deletion mouse model that was protected from kidney disease progression across three different disease models, without any known phenotypic changes under control conditions (Kolligundla et al., 2025)
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This may be partly due to lower total antioxidant activity in these patients, particularly the decrease in SOD and CAT activities (24, 25)
Comprehensive toxicology evaluations reveal that the compound does not affect food intake, body temperature regulation, or basic physiological parameters at effective doses in animal models, indicating that its metabolic effects occur through specific pathway modulation rather than general physiological disruption
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