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Epithalon also seemed to inhibit the activity of Caspase-3, a crucial enzyme believed to facilitate apoptosis (programmed cell death)
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Claims with weak, preliminary, preclinical, or unsupported evidence are identified as such rather than presented as established medical benefit
Its function is to inhibit the NNMT enzyme to enhance cellular energy and encourage fat cells to burn, rather than store, energy
Mitochondrial function, a critical aspect of oocyte viability, appeared to be also positively affected by Epithalon
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