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A few reports using traditional chemotherapeutics, e.g., carboplatin and etoposide, showed low response rates and no survival benefit [14]

Retatrutide works through simultaneous activation of three metabolic receptors: GLP-1 (which reduces appetite and improves insulin secretion), GIP (which enhances insulin sensitivity and influences fat storage), and glucagon (which increases energy expenditure and promotes hepatic fat oxidation)

Additionally, its insulin-independent action makes it useful in patients with insulin resistance or beta-cell dysfunction