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It has been shown that the neuroprotective effect of QCN against focal cerebral ischemia is mediated by activating PI3K/AKT signaling pathway and tropomyosin receptor kinase B (TrkB) receptor in rat model (Yao et al
Numerous mitotic figures are present
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After injury, endothelial cells participate in clearing myelin debris, which can induce additional damage to TJs and gap junctions
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