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Neurological research has explored its effects in models of traumatic brain injury and spinal cord compression, with several studies reporting improved functional recovery versus vehicle controls

Such a setup would precisely delineate the NSC-derived paracrine factors (e.g., BDNF, VEGF [18, 32], TGF-) responsible for suppressing microglial activation and promoting an anti-inflammatory phenotype, independently of in vivo complexities

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The secret often lies in blood flow and cellular communication

102 p53 also binds to several antiapoptotic proteins, including Bcl-2, Mcl-1, and Bcl-xL, indirectly inducing apoptosis