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The table below summarizes the clinical situations where each marker is preferred

Proposed mechanisms include upregulation of growth factor receptors, stimulation of fibroblast migration and collagen synthesis, and promotion of angiogenesis at injury sites

These effects are well-documented across multiple animal models of intestinal injury, ulcerative colitis, and inflammatory bowel disease

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Phosphoric acid reflects phosphate level, an essential mineral for bones, energy storage in ATP, and many cell functions, shaped by diet and vitamin D