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30 , 129142.e4 (2019)
Acquired resistance often originates from preexisting heterogeneity within the tumor and is further shaped by continuous diversification during therapy, enabling certain subclones to survive therapeutic pressure and eventually emerge as dominate resistant phenotypes
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Community research-use guides suggest a subcutaneous range of about 100200 mcg (overall roughly 50200 mcg), reconstituted with bacteriostatic water only [12]
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