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It promotes angiogenesis at the injury site, activates fibroblasts, enhances growth hormone receptor expression in tendon cells, and modulates inflammation
The increases in beta-hydroxybutyrate (78-181%) confirmed that the fatty acid oxidation mechanism was driving these changes at the cellular level
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Illuminating the impact of N-terminal acetylation: From protein to physiology
This preserves motility and decreases fragmented DNA rates, boosting fertilization potential
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