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Similar content being viewed by others Introduction Therapeutic resistance remains one of the most formidable challenges in oncology, limiting the long-term efficacy of anticancer therapies and contributing substantially to disease relapse and poor prognosis
No controlled human data exists for FOXO4-DRI
Studies with mutants and transgenic plants over- or under-expressing enzymes or metabolites of the AsA-GSH pathway have proved very well the co-relation between this pathway and stress tolerance (Gill and Tuteja, Each enzyme of the AsA-GSH pathway has various subcellular isoforms, which differ from each other with respect to their spatial and temporal expression
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Or And if so, how does one balance or even evaluate the balance between the two apparently contradictory effects
