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In mammalian systems, a subpopulation of caspase-3 in the mitochondria is constitutively S -nitrosylated and, as such, is inhibited, and mitochondrial Trx2 has been involved in this S -nitrosylation (Mitchell and Marletta, 2005), although the exact intramolecular mechanism of Trx denitrosylation remains ill-defined, as does the denitrosylation target specificity (Benhar et al., 2009)
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2,3 The accumulation of these errors raises aneuploidy rates and is widely recognised as the single biggest contributor to lower implantation rates and higher miscarriage rates after 35
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