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Oxidative stress in turn triggers hepatocyte injury and release of damage-associated molecular patterns (DAMPs), which activate Kupffer cells and recruit inflammatory cells through nuclear factor-kappa B (NF-B) signalling and inflammasome pathways (eg., NLRP3)
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5e,f), suggesting that SyeA is truncated during in vivo processing or fragmented during sample preparation
What Are the Synergistic Benefits of Stacking DSIP Epithalon Peptides
Dec 2013;59(4):329-340
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