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NHK is encoded by a nonsense mutation of 1-antitrpysin that results in a truncated product which is retained in the ER and undergoes ER-associated degradation, resulting in ER stress in diverse cell types (Nagasawa, Higashi, Hosokawa, Kaufman, & Nagata, 2007

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In Phase-I HSB, supplementation with 1% or 2% glycine increased ( P 0.05) in these variables between the 1% and 2% glycine groups

Anstatt supraphysiologische Spiegel zu erzwingen, wirkt es innerhalb des krpereigenen Hormonsystems