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69 In gastric cancer cells, the absence of CDO1 contributed to restore cellular glutathione levels, to prevent ROS and lipid peroxidation, and to promote resistance to ferroptosis, a type of programmed cell death dependent on iron and associated with lipid peroxide accumulation
j , Uninfected M treated with KL1 (10 or 40 M) were assessed for viability (CellTiter-Fluor
Reconstitute: Add 3.0 mL bacteriostatic water to the 10 mg vial ~ 3.33 mg/mL total (1.67 mg/mL each peptide)
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The sequence is Trp-Ala-Gly-Gly-Asp-Ala-Ser-Gly-Glu, an unusually acidic nonapeptide
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