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(2008) characterized the role of protein S-glutathionylation as a redox-sensitive post-translational modification, establishing glutathione as a signaling molecule beyond its antioxidant function [3]
[15] [16] In the brain, GLP-1 receptor activation has been linked with neurotrophic effects including neurogenesis [17] [18] and neuroprotective effects including reduced necrotic [19] and apoptotic [20] [19] signaling, cell death, [21] [22] and dysfunctions
Among the vitamins examined, VK, VQ, VU, VD, VB1, VB6, VB12, and VE could inhibit the activation of TLR signaling pathway, and VD, VB1, VB6, VB12, VE, and VC could inhibit the activation of NLR signaling pathway
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