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Cancer Cell 40 , 277288.e3 (2022)
After being activated by insulin signaling, Akt inactivates AS160 (GTPase-activating protein [GAP] AKT substrate of 160 kDa, also known as TBC1D4), which activates small Rab GTPase protein switches that control vesicle trafficking [10]
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Synthesis of ketone bodies may be initially increased in MASLD as a mechanism to safely manage excess triglyceridederived acetylCoA via nonoxidative disposal to limit oxidative stress, a mechanism which becomes disturbed as MASLD progresses (Post et al
A.IkonomopoulosI.BannisterA
