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2.2 Iron overload, deficiency, and IBD pathogenesis In the intestinal microenvironment of IBD, iron overload or functional iron deficiency not only serves as a source of oxidative stress but also acts as a critical factor triggering ferroptosis in intestinal epithelial cells
Similarly, redox potential was significantly reduced in APAP-treated animals (-206.77 0.65 mV for vehicle control vs -184.52 3.34 mv for APAP treated) consistent with increased oxidant exposure
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Importantly, the treatment with NP4 showed the strongest therapeutic effect (individual tumor growth inhibition curves: Fig
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