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Similarly, semaglutide ameliorates doxorubicin-induced cardiotoxicity by reducing BNIP3-mediated mitochondrial dysfunction through the PI3K/AKT pathway, enhancing mitochondrial function and reducing cardiac injury (Li et al., 2024), potentiating its clinical application in cardiovascular diseases

They received semaglutide 0.5 mg weekly

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Interestingly, long-acting L-DOPA treatment also results in an enhanced biosynthesis of A 2A Rs in patients with PD-induced dyskinesias, especially in brain networks involving the striatum and substantia nigra [24]

The scope of the problem is not a one-off event