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How does the selective binding of GLP-1 receptor agonists to pancreatic -cells influence their role in regulating insulin secretion and glucagon release

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[4] enhanced cell migration, where MSCs respond to chemotactic signals from the injury site and pro-migratory factors secreted by themselves, thereby homing to the skin wound area and participating in wound repair alongside KCs, macrophages, and endothelial cells migrating to the wound tissue [125, 126]

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